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Contech Enterprises Inc tmt chemicals
Tmt Chemicals, supplied by Contech Enterprises Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/tmt+chemical/tmt+chemical/pm32973323-233-9-21
Average 90 stars, based on 1 article reviews
tmt chemicals - by Bioz Stars, 2026-09
90/100 stars

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Related Articles

Comparison:

Article Title: Where is the TMT? GC-MS analyses of fox feces and behavioral responses of rats to fear-inducing odors.
Article Snippet: For the purpose of comparison, 11 of the 24 fox samples had TMT added in varying concentrations (20, 100, 200, and 800 nmol TMT to 0.4 g fox feces, thus 50, 250, 500, and 2000 nmol of TMT per g of fox feces) using serial dilutions in ethanol of commercially obtained TMT (trimethylthiazoline, also known as 2,5-dihydro-2,4,5-trimethylthiazoline; 2,3,5-trimethyl-3-thiazoline; or 2,4,5-trimethyl-4,5-dihydro-1,3-thiazole; molecular formula C6H11NS; molecular mass 129.223 g/mol; CAS no. 4145-93-1; Contech, British Columbia, Canada).

Article Title: Endogenously Released Neuropeptide Y Suppresses Hippocampal Short-Term Facilitation and Is Impaired by Stress-Induced Anxiety
Article Snippet: Predator scent stress was induced using two exposures to TMT (2,5-dihydro-2,4,5-trimethylthiazoline; Contech), a chemical from fox feces that elicits a fear response in rodents ( Fendt and Endres, 2008 ; Rosen et al., 2015 ).

Article Title: Endogenously Released Neuropeptide Y Suppresses Hippocampal Short-Term Facilitation and Is Impaired by Stress-Induced Anxiety
Article Snippet: Predator scent stress was induced using two exposures to TMT (2,5-dihydro-2,4,5-trimethylthiazoline; Contech), a chemical from fox feces that elicits a fear response in rodents (Fendt and Endres, 2008; Rosen et al., 2015).

Article Title: Cognitive Deficits and Inflammatory Response Resulting from Mild-to-Moderate Traumatic Brain Injury in Rats Are Exacerbated by Repeated Pre-Exposure to an Innate Stress Stimulus.
Article Snippet: Traumatic brain injury (TBI) is common in both military and civilian populations, and often results in neurobehavioral sequelae that impair quality of life in both patients and their family.. Although individuals who are chronically exposed to stress are more likely to experience TBI, it is still unknown whether pre-injury stress influences the outcome after TBI.. The present study tested whether behavioral and cognitive long-term outcome after TBI in rats is affected by prior exposure to an innate stress stimulus.

Multiplex sample analysis:

Article Title: Where is the TMT? GC-MS analyses of fox feces and behavioral responses of rats to fear-inducing odors.
Article Snippet: For the purpose of comparison, 11 of the 24 fox samples had TMT added in varying concentrations (20, 100, 200, and 800 nmol TMT to 0.4 g fox feces, thus 50, 250, 500, and 2000 nmol of TMT per g of fox feces) using serial dilutions in ethanol of commercially obtained TMT (trimethylthiazoline, also known as 2,5-dihydro-2,4,5-trimethylthiazoline; 2,3,5-trimethyl-3-thiazoline; or 2,4,5-trimethyl-4,5-dihydro-1,3-thiazole; molecular formula C6H11NS; molecular mass 129.223 g/mol; CAS no. 4145-93-1; Contech, British Columbia, Canada).

Article Title: Endogenously Released Neuropeptide Y Suppresses Hippocampal Short-Term Facilitation and Is Impaired by Stress-Induced Anxiety
Article Snippet: Predator scent stress was induced using two exposures to TMT (2,5-dihydro-2,4,5-trimethylthiazoline; Contech), a chemical from fox feces that elicits a fear response in rodents ( Fendt and Endres, 2008 ; Rosen et al., 2015 ).

Article Title: Endogenously Released Neuropeptide Y Suppresses Hippocampal Short-Term Facilitation and Is Impaired by Stress-Induced Anxiety
Article Snippet: Predator scent stress was induced using two exposures to TMT (2,5-dihydro-2,4,5-trimethylthiazoline; Contech), a chemical from fox feces that elicits a fear response in rodents (Fendt and Endres, 2008; Rosen et al., 2015).

Article Title: Cognitive Deficits and Inflammatory Response Resulting from Mild-to-Moderate Traumatic Brain Injury in Rats Are Exacerbated by Repeated Pre-Exposure to an Innate Stress Stimulus.
Article Snippet: Traumatic brain injury (TBI) is common in both military and civilian populations, and often results in neurobehavioral sequelae that impair quality of life in both patients and their family.. Although individuals who are chronically exposed to stress are more likely to experience TBI, it is still unknown whether pre-injury stress influences the outcome after TBI.. The present study tested whether behavioral and cognitive long-term outcome after TBI in rats is affected by prior exposure to an innate stress stimulus.



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